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If you’ve ever witnessed someone suffer a stroke, you understand the humbling nature of this disease. It can reduce the mightiest human being to an immobile, helpless creature. Impairment of crucial functions like speech, walking, and control of bowel and bladder can wrench control from the body in a moment. Even perpetually youthful TV personality Dick Clark was struck down by stroke at age 75, despite the outward appearance of perfect health. Clark’s stroke resulted in a six-week hospital stay and, judging from fragmented reports, significant disability. Stroke can be like a devastating fire that strikes without warning, leaving only smoldering rubble. Stroke can so ravage basic bodily functions that often all you can hope for is to regain a portion through rehabilitation. The disease process that underlies stroke requires decades—30 or 40 years—to develop. With that much lead time, why aren’t we better able to detect or stop this crippling disease? The truth is that we are able to predict many, if not most, strokes. Advances in imaging technology allow detection of atherosclerotic plaque that cause stroke years before it becomes a threat. Progress in deciphering the causes of stroke has also leapt forward. Unfortunately, your neighborhood physician still focuses on diagnosing the crisis rather than anticipating it. Physicians prefer to deal with catastrophes and are just not that interested in prevention. Most physicians ask: “Is it time to operate or not?” The medical community obsesses over procedures like carotid endarterectomy (surgical removal of plaque) or carotid stents. Even when a person is afforded the warnings of a “mini-stroke”, or transient ischemic attack (TIA), little more is done once it’s determined that surgery is not necessary—even though this person has high risk for future stroke (50% over 10 years). Let’s flip-flop this approach to stroke. Procedures represent a failure of prevention! Where do strokes come from? Stroke develops when some portion of the brain is deprived of blood. This usually results from a tiny bit of debris that dislodges from an atherosclerotic plaque along the walls of an artery (the same sort that accumulates in coronaries causing heart attack). The sources of debris have been a subject of controversy, but new imaging technologies have settled the question. Any blood vessel that leads from the heart to the brain can be a source. The two carotid arteries on both sides of your neck are a frequent source, as these arteries are prone to develop plaque. (Our discussion will be confined to what are called thromboembolic, or ischemic, strokes, i.e, strokes that occur from plaque that fragments, sending debris to the brain, and will not include the far less common hemorrhagic strokes due to rupture of small vessels in the brain, nor will we discuss atrial fibrillation and other heart causes of stroke. The thromboembolic strokes we discuss cause around 88% of all strokes.) Over the last 10 years, the aorta has been recognized as another important source of stroke. The aorta is the main artery of the body whose branches go to the head, arms, and legs. Atherosclerotic plaque is a live tissue that, through poor diet, inactivity, high cholesterol, overweight, etc., grows and becomes progressively more unstable. At some point, plaque fragments. Little bits break away, traveling to the brain. Fractured plaque also exposes its deeper structures to flowing blood, triggering blood clot formation, which in turn can also fragment and go to the brain. Atherosclerotic plaque is a prerequisite for the most common causes of stroke. If the majority of strokes originate from plaque, why not measure plaque to determine if you’re at risk for stroke? How can we easily, safely, and accurately measure plaque in the carotid arteries and aorta? And if plaque can be measured, can it be shrunk or inactivated to reduce or eliminate risk for stroke? How can plaque be measured? Just 20 years ago, the only practical method of identifying plaque in the carotids or aorta was through angiography, requiring catheters inserted into the body to inject x-ray dye. Angiography was impractical as a screening measure. CT scanning and magnetic resonance imaging (MRI) are emerging as exciting methods of imaging both carotids and aorta. Unfortunately, most centers and physicians are much more focused on the diagnostic uses of these technologies for people who have already suffered stroke or other catastrophe, and application of these devices for preventive uses is still evolving. One exception is when aortic calcification or aortic enlargement is incidentally noted on the increasingly popular CT heart scans; this is an important finding that can signal presence of aortic plaque. The one test that is widely available and can be performed in just about any center is carotid ultrasound. It’s simple, painless, and precise. Two basic observations can be made: 1. Plaque detection—Atherosclerotic plaque can be clearly visualized. If plaque blocks more than 70% of the diameter of the vessel, or if there are “soft” (unstable) elements in plaque, then stroke risk may be high enough to justify surgery or stents. However, if there are plaques that are less severe, substantial risk for stroke may still be present that can be reduced with preventive measures. 2. Carotid intimal-medial thickness—This is a measure of the thickness of the lining of the carotid artery in areas not involved by plaque, but often precedes the development of mature plaque. Carotid intimal-medial thickness also provides an index of body-wide potential for atherosclerotic plaque that can place you at risk for stroke. The aorta, for instance, cannot be well imaged by surface ultrasound but can still be a source for stroke. Increased carotid intimal-medial thickness and carotid plaque are closely associated with likelihood of aortic plaque. The Rotterdam Study of 4000 participants demonstrated that if carotid intimal-medial thickness is greater than normal (1.0 mm), then you can be at risk for stroke (and heart attack), even if no carotid plaques are detected. Carotid ultrasound is the one test you should consider that provides the most information with least effort. Ultrasound is harmless, painless, and can be obtained just about anywhere. Even if your doctor disagrees with your request for a carotid ultrasound, an increasing number of mobile services are popping up nationwide that make this test available for around $100. One important point: many scanners and interpreters will only report whether plaque is present or not. While this is important information, you should request that the carotid-intimal medial thickness be made as well. Not all centers can make this simple measure (because of software requirements), but it doesn’t hurt to try. Any amount of carotid plaque is reason to follow a preventive program, even if the plaque is insufficient to justify surgery. Can plaque be reduced? Can we shrink plaque in carotid arteries and aorta and thereby reduce, perhaps eliminate, these sources of stroke? That question is gaining momentum as effective therapies become available that pack real punch for reducing plaque. Study after study has now documented that plaque can be reduced and, with it, risk for stroke. Reduction in plaque of 10–20% is possible within a year or two. Let’s consider the most potent influences on carotid and aortic plaque growth that need to be considered in a plaque-reducing program. (I assume that you are a non-smoker—if you are a smoker, you first need to concentrate on quitting.) Hypertension Considerable experience documents the power of blood pressure-lowering for prevention of stroke. The most recently updated guidelines, the JNC–VII, recommends a blood pressure of 407 mg/dl heightens stroke risk six-fold. C-reactive protein (CRP) This measure of inflammation is proving to be a useful marker for identifying people at risk for stroke, with increased risk beginning at a level of 0.5 mg/l. High CRP also predicts more rapidly growing carotid plaque. Homocysteine Homocysteine is an important marker of increased likelihood of both carotid and aortic plaque, as well as stroke. In 1997, the European Concerted Action Project reported more than a doubling of stroke when homocysteine levels exceeded 12 mol/l. As homocysteine increases to 20 μmol/l, risk for stroke and heart attack increases an amazing 10-fold over that at a level of 9 μmol/l. Asymmetric dimethylarginine (ADMA) ADMA is recently discovered amino acid whose blood levels can skyrocket up to 10-fold in the presence of hypertension, metabolic syndrome, diabetes, high cholesterol and triglycerides, obesity, and high homocysteine levels. ADMA blocks the action of the amino acid, l-arginine. This mimicry reduces the availability of nitric oxide, a powerful dilator and protector of arteries. ADMA levels in the top 10% predict a six-fold heightened risk for future stroke, and ADMA levels in people with strokes are double that in other people. A carotid ultrasound study in 116 subjects showed that higher blood levels of ADMA are associated with more severe carotid plaque. Because of ADMA’s shared role across a variety of abnormal conditions, correction or blocking the action of ADMA has been suggested as a unique therapeutic tool to reduce stroke risk. Cholesterol Data suggest that lowering cholesterol with statin cholesterol-lowering drugs slows carotid plaque growth and reduce stroke risk approximately 22%. An interesting study from the Cardiovascular Institute at Mt. Sinai School of Medicine in New York using the precise measuring ability of MRI of the carotids and thoracic aorta showed an impressive 20% regression of plaque area with simvastatin (Zocor®) taken for two years. Although guidelines for cholesterol treatment recommend reduction of LDL cholesterol to 100 mg/dl in high-risk persons, a report from the Walter Reed Army Medical Center in Washington, DC, showed that carotid plaque was more effectively reduced when LDL cholesterol of 70 mg/dl or lower was achieved with statin cholesterol drugs. Lower LDL cholesterol may, therefore, be better. Treatment Strategies to Reduce Carotid and Aortic Plaque The essential question: How do we reduce carotid and aortic plaque? If we make this the focus of our efforts, many pieces begin to fall into place. If you’ve had any measure of carotid or aortic plaque such as a carotid ultrasound or aortic calcification on a CT heart scan, you know that you’re at increased risk for stroke. You also have a baseline for future comparison to gauge whether your program is working or not. Because most people have not one but several causes of carotid and aortic plaque, there is no one single treatment that effectively eliminates risk for stroke. Instead, most people require a comprehensive program of healthy diet, exercise, supplements, and medication when indicated. Here, we focus on the nutritional supplements that can be critical components of your plaque-reduction program. Fish oil Fish oil is a cornerstone of your stroke prevention program. Epidemiological observations suggest a strong relationship of fish intake and reduction of stroke risk. Carotid ultrasound studies demonstrate less carotid plaque with greater intakes of fish. A cleverly designed University of Southampton study made the fascinating observation that fish oil transforms the structure of carotid plaque. 150 people with severe carotid plaque scheduled for carotid endarterectomy (surgical removal of the plaque) were given fish oil, sunflower oil, or no treatment over several months while waiting for their procedure. (Delays in the British health system permitted this unique design.) Plaque was removed at surgery and examined. Participants taking fish oil had reduced inflammation in plaque and thicker tissue covering the fatty core, markers of more stable plaque. Those taking sunflower oil or no treatment had unstable plaques with greater inflammation and thinner, less sturdy covering tissue. This suggests that fish oil stabilizes carotid plaque, making it less likely to rupture and fragment. A standard capsule of fish oil (containing 300 mg of EPA + DHA) contains the same amount of omega-3s as a 3 oz serving of cod or halibut; three capsules (900 mg DHA + EPA) contain the equivalent of a serving of farm-raised salmon. The dose that seems to provide greatest protection from stroke, lowers triglycerides (that form abnormal lipoproteins; see above), and reduces fibrinogen, is four capsules per day (1200 mg EPA + DHA). Coenzyme Q10 (CoQ10) Although there are no data specifically addressing whether CoQ10 reduces plaque, it is a marvelously effective way to reduce blood pressure, one of the crucial factors causing carotid and aortic plaque growth. A pooled analysis of eight studies showed that, on average, CoQ10 in daily doses of 50–200 mg reduced systolic blood pressure by 16 mm Hg, diastolic pressure by 10 mm Hg. Data suggest that CoQ10 can reverse abnormal heart muscle thickening (hypertrophy), another manifestation of high blood pressure, strongly suggesting that CoQ10 has benefits beyond just reducing pressure. Supplements to correct the metabolic syndrome Weight loss is, without question, the most immediate and direct path to correction of this dangerous pre-diabetic condition. A drop of even 10–20 lbs yields improvements across the board: increased sensitivity to insulin, increased HDL, and reductions in triglycerides, CRP, fibrinogen, small LDL particles, and blood pressure. Diet and exercise are fundamental components of an effort to lose weight; low carbohydrate or reduced glycemic index diets (e.g., South Beach or Mediterranean) rich in fibers are clearly effective. Several supplements can amplify weight-reduction efforts and be useful adjuncts to your lifestyle program. Among them: White bean extract White bean extract blocks intestinal absorption of carbohydrates by 66%. 1500 mg twice a day with meals yields, on average, 3–7 lbs of weight loss in the first month of use. The only side-effect is excessive gas, due to unabsorbed starches. Glucomannan This unique fiber taken prior to meals absorbs many times its weight in water and thereby fills your stomach. You consequently take in less food. Most people lose around four lbs per month using 1500 mg prior to each meal. Interestingly, glucomannan also blunts the rise in blood sugar after meals, an effect that, by itself, may lead to weight loss. Be sure to take with plenty of water. DHEA This adrenal hormone is key to maintaining physical stamina, mood, muscle mass in men, and libido in women. A recent randomized, placebo-controlled study at Washington University in 56 subjects showed a 13% decline in abdominal fat (fat that drives resistance to insulin) measured by MRI with 50 mg of DHEA per day at bedtime, along with improved sugar control and lower insulin levels. Pectin, beta-glucan Pectin is the soluble fiber in citrus rinds, green vegetables, and apples, also available as a supplement. Beta-glucan is the soluble fiber of oats and is also available as a supplement. Both are wonderful fibers that provide feelings of fullness, lower cholesterol, slow release of sugars, and can yield modest weight reduction. A USC study in 573 subjects using carotid ultrasound showed that greater intake of healthy fibers like pectin and beta-glucan is associated with less carotid plaque growth. Folic acid, vitamins B6 and B12 Dr. Daniel Hackam at the Stroke Prevention and Atherosclerosis Research Centre in Ontario conducted a study using carotid ultrasound in 101 participants treated with folic acid 2.5 mg, vitamin B6 25 mg, and B12 250 mcg per day. Treatment resulted in plaque reduction, especially when homocysteine levels exceeded 14μmol/l at the start, compared to untreated participants who experienced substantial plaque growth. An attempt to clarify the role of homocysteine treatment was made through a National Institute of Health-sponsored study of stroke prevention. 3680 participants with a prior history of stroke were enrolled and given either a “low-dose” (20 mcg folic acid, 0.2 mg B6, 6 mcg B12) or a “high-dose” (2.5 mg folic acid, 25 mg B6, 400 mcg B12) regimen. Although starting homocysteine levels showed a graded association with stroke risk (higher homocysteine levels predicted greater stroke risk), the treatment groups experienced, on average, only a 2 μmol drop in homocysteine levels and no reduction in stroke risk over two years. The study investigators as well as critics have suggested that the study failed due to an insufficient treatment period and that the doses were too low. (The doses we use in our plaque reduction program are folic acid 2.5–5.0 mg, B6 50–100 mg, B12 1000–2500 mcg.) L-arginine L-arginine can be used to overpower the adverse effects of ADMA. L-arginine is emerging as an important carotid plaque-reversing tool. Early reports in animals showed that l-arginine completely halted growth of aortic plaque, and did so more effectively than lovastatin (a cholesterol-lowering drug). In humans, L-arginine reduces blood pressure, abnormal constriction of carotid and coronary arteries, blocks entry of inflammatory cells into plaque, increases sensitivity to insulin, and heightens exercise capacity. Following coronary angioplasty or stent placement, l-arginine results in up to 36% reduction in plaque growth. The average American takes in 5400 mg of l-arginine through food every day. Supplementing with doses of 3000–12,000 mg per day has proven useful to correct many of these phenomena. (We use a dose of 6000 mg of l-arginine powder, twice a day on an empty stomach, dissolved in water, for our plaque regression program.) Does this result in a reduction of stroke risk? The emerging data suggest that l-arginine is likely to exert a powerful plaque-reducing and stroke-preventing benefit, but we await more clinical trial data. Conclusion Reducing stroke risk by reversing carotid and aortic plaque is becoming an everyday reality, with better tools becoming available. To know whether you’re at risk, the best and most available imaging tool is carotid ultrasound, aiming to identify intimal-medial thickness >1.0 mm, or carotid plaque. Any degree of calcification of the aorta, such as on a CT heart scan, is another useful measure of risk. Treatment to reduce risk is multi-faceted but is based on examining all your sources of risk, including metabolic syndrome, small LDL, lipoprotein(a), and C-reactive protein. Fish oil is the one absolutely crucial ingredient in any stroke prevention program. Other supplements can be used in a targeted fashion, depending on the causes identified for your carotid or aortic plaque. Ideally, repeat scanning of your carotids should be done sometime after your program has begun to assess whether you’ve successfully achieved reversal of plaque growth. manual pnis enlargement exercise vigrx penis enlagement pill vimax natural penis enlargement and lengthening best penile enlargment pills natural penis enlarement exercise guide to pennis enlargement compare pennis enlargement pills vimax enlargement manhattan penis

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Pelvic pain is a widespread chronic pain condition that affects the lives of men and women all across the world. But most people don't realize how big the problem is because those who suffer don't talk about it. Why? Would you talk about your pain if it involved rectal pain or vaginal/penis pain to your pals? My guess is no. You would keep it a secret and just try to "brush it under the rug" so to speak. There are so many people that suffer and just accept their pain which is sad. But change is coming... In the past few years, pelvic pain is getting much more recognition. Awareness is beginning to grow and people are coming out and talking. So you may be asking yourself, do I suffer from chronic pelvic pain? In a nutshell, chronic pelvic pain conditions usually consist of urinary pain, rectal pain, muscle spasms or tightness in the pelvic muscles, and usually there is no sign of any major abnormalities. That is what makes the condition so tough, is that there is nothing actually found to be wrong with the sufferer. Previously, most doctors would prescribe rounds of antibiotics or pain killers to take care of the pain. In most of the cases, there wasn't even any sign of an infection. Doctors would just prescribe what they thought might work. Overtime, this method didn't work. Today, there is a whole new approach to how pelvic pain is treated. One of the popular methods that has had good success is The Pelvic Pain Solution, an ebook written by a sufferer that beat pelvic pain (see www.redmaven.com )It is highly recommended if you suffer from chronic pelvic pain. The whole approach focuses on the body and the mind to beat pelvic pain. It is a more holistic approach that doesn't involve antibiotics or other invasive procedures. Finally, people that suffer from chronic pelvic pain conditions such as prostatitis, vulvodynia, interstitial cystitis, levator ani syndrome, etc. can have hope. The old methods involving pointless procedures (dilations, pain killers, antibiotics, invasive procedures) are about to bite the dust. There is a new turn to a more realistic treatment that puts the sufferer in charge of healing themselves...instead of someone else trying to heal them. This is new breath of fresh air for people with pelvic pain! For more information on chronic pelvic pain conditions, visit: www.chronic-pelvic-pain.redmaven.com do penis enlarement pills work vimax free penis enlargement pills penis enlarement before and after picture free natural penis enlargement penis enlarement pills natural pnis enlargement penile enlargement before and after penis enlarement excercises natural penis enlargement and lengthening

Brits moving permanently to Bulgaria It has been reported that many Brits are falling for Bulgaria 's charms with a growing number choosing to move there permanently. In recent years investment has been the main driving force behind the number of Brits buying property in the country but many have decided to buy and live outside the Sunny Beach area and a few miles inland as well as other village areas around the country. There are a number of factors for those choosing to move but the low cost of living and the seemingly peaceful surrounds are the most likely reasons so many Brits are making the move. Many have also been able to rent out there properties back in the UK , and live very comfortably off the rental income in Bulgaria . As more companies relocate to Bulgaria to take advantage of the low wages and to strategically place themselves for the countries' expected accession to the EU in 2007, many British employees are expected to relocate with their employers. Tourist areas have seen recent year-on-year growth of between 25% up to as much as 100% however those opting to live in to move permanently prefer to be based inland. Many Brits are buying around the Veliko Turnovo region with home prices starting as low as GBP 10,000. There are still cheaper homes available however many will require additional funds to provide for renovations. The move is been made by both retirees who find they can make their pension stretch a lot further and younger people looking for new business opportunities and finding it difficult to get on the property ladder in the UK. EU Final word on Bulgaria to be known soon Oli Rehn, European Enlargement Commissioner is soon expected to present his report on Bulgaria Romania's accession to the EU in January 2007. It is generally believed that the planned memberships will go ahead however the accession treaty does contain a safeguard clause allowing membership to be postponed by one year if further reforms in each country are deemed necessary. safe pnis enlargement penis enhancement pic vig rx hoax pennis enlargement traction device penis enargement pic penile enlargment surgeries penis enlargment review penile enlargement program natural penis enlargement and lengthening

Do you know what exactly is special about Levitra? It is Vardenafil hydrochloride, a strong molecule with a weight of 488.604 g/mol. As a PDE5 inhibitor, Vardenafil is closely related in both function and marketing to Sildenafil and Tadalafil though; it has a comparatively short effective time with less instances of failure. Those who have Levitra experience, have pointed out that unlike other ED pills, Levitra has worked for them on the very first attempt. Let’s find out the actual selling points of the ED drug Levitra over its competitors. The main ingredient of Levitra, Vardenafil hydrochloride is a potent molecule which requires smaller dosage to get into full action. The scientists refer to the molecule as far more stronger than Sildenafil and Tadalafil. Levitra works wonders for men who had unsatisfactory or failed experience with Viagra. This statement has been confirmed time and again by the result of different research studies. Vardenafil hydrochloride in Levitra is a boon to the diabetes patients, who are at a greater risk of erectile dysfunction due to uncontrolled sugar in blood that impedes the blood flow to the penis. Levitra has proved to be effective even in tight situations like high blood pressure as well as diabetes type2 when other ED drugs have failed to work. Levitra’s highest dose of 20 mg is equal to 100 mg maximum dose of Viagra. Now, check out the lowest dose for Viagra, 12.5 mg and compare it to 2.5 mg pill of Levitra, its lowest dose. Those, who have been hit below the belt, are found to experience fewer side effects compared to other available PDE5 inhibitors. The mild and most common side effects are headaches, nasal congestion, flushing. What is more, with Levitra, you need not worry about your eyes as it has no detrimental side effects on vision. There is another add-on benefit with Levitra; it is less sensitive to food and alcohol, again, giving you ample chance for a romantic dinner with your ladylove while getting ready for the right moment. The ED Market Each of the PDE5 inhibitors will have it’s share in the erectile dysfunction market. It seems that Levitra has a promising future ahead, because, Vardenafil hydrochloride is strong enough to give an erection to the diabetics, who are the most easy victims of impotence, therefore considered as a substantial chunk of ED market. The ever increasing market of erectile dysfunction is experiencing a head-on competition within the trio. For me, the ED market scenario is quite clear! Viagra, Levitra & Cialis, all of them have something special to offer. If Viagra has the longest track record, Cialis has 36 hours of action to offer, while Levitra has come up with effectiveness and gives strong erection to diabetics even with smaller doses. The ED market giant remains Viagra though, owing to its consistency in performance but Levitra wins in rigid situations like diabetes. Based on its advantages, Levitra has captured 50% of prescriptions for newly diagnosed erectile dysfunction patients. The MarketWatch report makes it almost clear as water, in spite of Viagra’s strong hold on the market, Bayer recorded a 16% advance in sales of its erectile dysfunction drug, to 73 million euros this year. The figures clearly show that Levitra will not wait for long to defeat its competitors. ___________________________________________________________________ pennis enlargement photo penis enlargement result penile enlargment excercises penis enlagement tool natural penis enhancement exercise penis enargement picture penis enlargment patch natural penis enlagement pills natural penis enlargement and lengthening

Unhappy with their breast size and how they look, more and more girls under the age of 18 are undergoing expensive and dangerous breast implant surgery. As reported by the American Society of Plastic Surgeons, 3,841 young women 18 or younger underwent breast augmentation, a 24 percent jump from 3,095, a 19 percent increase from 2002. From coast to coast, and especially prevalent in Texas and California, breast implants are fast becoming the most popular new fad among teens looking to improve their looks and become more noticeable to the opposite sex. 'Breasts are a fashion item,' says Dr. Garry Brody, professor of plastic surgery at the University of Southern California in Los Angeles. More and more teens regard this dangerous practice as nothing more than fun, willing to ignore the danger, side effects, and long term problems associated with breast implant surgery. Many teens regard the surgery as "just a fun thing to do," comparing it to having their hair or nails done, rather than the complicated, dangerous, and expensive surgical procedure it really is. Around holidays, spring break, graduation, and especially during the Christmas season, doctors begin to see the demand for implant surgery increase. Breast augmentation has become so popular among teens, that many parents are now offering the surgery as a Sweet 16 or high school graduation gift, sometimes paying upwards of $10,000 for breast enlargement. Even though in 2000 the Food and Drug Administration approved saline implants for women 18 or older, its recommendation is only advisory, because unlike medications, breast implants are not regulated by them. However, silicone-gel implants are not approved for marketing, available to women only through FDA approved clinical studies. Throwing caution to the wind, teens are unwilling to treat implant surgery with the seriousness it deserves, acknowledging the risks and permanent changes they're making to their bodies and appearance. And unfortunately those risks far outweigh the advantages to having larger, fuller breasts, especially at such a young age, when most teen's bodies aren’t even fully developed yet. Most teens don't want sensible advice thrown at them, though, especially if they're flat-chested girls still in the process of developing. Unfortunately at that age, teen girls have the worst self and body image. When they look through magazines and see voluptuous models with large, full breasts on every page, they want to look just like that, the consequences be damned. And there are always consequences to breast implant surgery, some serious, some not, but one thing is for sure, their bodies will be forever changed. And the saddest thing is that once the surgery is completed, it can't be undone. They're stuck with facing, what for some of these young girls, will end up being a lifetime of regret. Everything from severe asymmetry to life threatening complications and illness can occur with implant surgery. Other risks include complications from anesthesia, excessive bleeding and infection, to name just a few. Also, over time the implants can rupture or deflate, requiring more surgery, along with capsular contracture the tightening of scar tissue around the implant, a common complication following breast augmentation. Fortunately today there are other safer, all natural remedies, and alternatives to increasing breast size available on the market that can be just as effective, with none of the side effects or dangers involved with implant surgery. If they're willing to take the time to look around, women and teens can have the larger, fuller breasts they want, without risking their lives to have it.